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Camellia Oil or Tea Seed Oil (Camellia oleifera)

http://winvivo.com/files/botanicals/buddha%20fruit%20-%20momordica%20grosvenori%20-%20image.pngThe seeds of Camellia oleifera are pressed to yield camellia oil or tea seed oil.

Traditional Uses of Camellia Oil or Tea Seed Oil
(Camellia oleifera)

  • Tea seed oil contains 80% mono-unsaturated oleic acid and vitamin E, which resembles olive oil and grape seed oil in its excellent storage qualities and low content of saturated fat.
  • With its high smoke point (485°F), for thousands of years camellia oil has been the main cooking oil in southern part of China.
  • In Japan, Korea and China camellia oil is used extensively for making skin care products due to its excellent moisturizing and anti-wrinkle/scarring properties, as well as in hair care products to enhance shine and manageability of the hair.

Modern Research - Camellia Oil or Tea Seed Oil (Camellia oleifera)

  • Composed mainly of oleic acid (almost 82%, the highest among all natural oils), camellia oil is the most penetrative of all natural oils, capable of permeating deeply into the lower layers of the skin to greatly enhance the beneficial effects of our natural collagen and elastin.
  • Oleic acid is known as an excellent transdermal carrier of cell rebuilding nutrients and bioactive compounds into the skin to repair the damage caused by dryness, sun exposure and other factors. The result is restoration of the skin's elasticity, significant improvement in skin texture as well as elimination or remarkable reduction of fine lines and wrinkles.

Camellia oil provides the skin with the necessary essential fatty substances which are critical in the retention and enhancement of skin moisture. A lack of these fatty substances will impair the skin's ability to retain moisture. The composition of camellia oil is very similar to that of the fatty acids of human skin. As a result, it can be easily and effectively absorbed into the skin to work in synergy wit

Triacylglyceride, antioxidant and antimicrobial features of virgin Camellia oleifera, C. reticulata and C. sasanqua Oils.

Molecules. 2013, 18(4):4573-87.
Feás X, Estevinho LM, Salinero C, Vela P, Sainz MJ, Vázquez-Tato MP, Seijas JA.
Department of Organic Chemistry, Faculty of Sciences, University of Santiago de Compostela, E-27080 Lugo, Spain.

Virgin oils obtained from seeds of Camellia oleifera (CO), Camellia reticulata (CR) and Camellia sasanqua (CS) were studied for their triacylglyceride composition, antioxidant and antimicrobial activities. Levels of fatty acids determined by ¹H-nuclear magnetic resonance analysis were similar to those reported for olive oils (82.30%-84.47%; 5.69%-7.78%; 0.26%-0.41% and 8.04%-11.2%, for oleic, linoleic, linolenic and saturated acids, respectively). The CR oil showed the best antioxidant potential in the three in vitro models tested. With regard to EC₅₀ values (µg/mL), the order in DPPH radical-scavenging was CR (33.48) < CO (35.20) < CS (54.87). Effectiveness in reducing power was CR (2.81) < CO (3.09) < CS (5.32). IC₅₀ for LPO inhibition were 0.37, 0.52 and 0.75 µg/mL for CR, CO and CS, respectively. All the oils showed antimicrobial activity, and exhibited different selectivity and MICs for each microorganism tested (E. coli, B. cereus and C. albicans). B. cereus was the less sensitive species (MIC: 52.083 ± 18.042 for CO; 41.667 ± 18.042 for CR; 104.167 ± 36.084 for CS mg/mL) and the E. coli was the most sensitive to camellia oil's effect. The standard gentamicin presented higher MIC for E. coli (4.2) than the CR (MIC= 2.6) and CO (MIC = 3.9) oils.

Antioxidant activity and bioactive compounds of tea seed (Camellia oleifera Abel.) oil.
J Agric Food Chem. 2006 Feb 8; 54(3):779-84.
Lee CP
, Yen GC.
Department of Food Science and Biotechnology, National Chung Hsing University, 250 Kuokuang Road, Taichung 40227, Taiwan, Republic of China.
Abstract

The oil of tea seed (Camellia oleifera Abel.) is used extensively in China as cooking oil. The objectives of this study were to investigate the antioxidant activity of tea seed oil and its active compounds. Of the five solvent extracts, methanol extract of tea seed oil exhibited the highest yield and the strongest antioxidant activity as determined by DPPH scavenging activity and Trolox equivalent antioxidant capacity (TEAC). Two peaks separated from the methanol extract by HPLC contributed the most significant antioxidant activity. These two peaks were further identified as sesamin and a novel compound: 2,5-bis-benzo[1,3]dioxol-5-yl-tetrahydro-furo [3,4-d][1,3]dioxine (named compound B) by UV absorption and characterized by MS, IR, 1H NMR, and 13C NMR techniques. Sesamin and compound B decreased H2O2-mediated formation of reactive oxygen species in red blood cells (RBCs), inhibited RBCs hemolysis induced by AAPH, and increased the lag time of conjugated dienes formation in human low-density lipoprotein. The results indicate that both compounds isolated from tea seed oil exhibit remarkable antioxidant activity. Apart from the traditional pharmacological effects of Camellia oleifera, the oil of tea seed may also act as a prophylactic agent to prevent free radical related diseases.

Sasanquasaponin protects rat cardiomyocytes against oxidative stress induced by anoxia-reoxygenation injury.
Eur J Pharmacol.
 2007 Dec 1; 575(1-3):21-7. Epub 2007 Jul 31.
Chen HP
, He M, Huang QR, Liu D, Huang M.
Institute of Clinical Pharmacology, School of Pharmaceutical Sciences, Sun Yat-Sen University, Guangzhou, PR China.
Abstract

Reactive oxygen species can play an important role in the pathogenesis of anoxia-reoxygenation injury. Sasanquasaponin (SQS) is a biologically active ingredient extracted from the Chinese medicinal plant Camellia oleifera Abel. Some studies have shown that SQS possesses potent antioxidant activities. However, it has not been elucidated whether SQS diminishes reactive oxygen species stress induced by anoxia-reoxygenation injury in cardiomyocytes. In this work, neonatal rat cardiomyocytes pretreated with the test compound were subjected to anoxia-reoxygenation. The extent of cellular damage was accessed by cell viability and the amount of released lactate dehydrogenase (LDH). Superoxide dismutase, catalase and glutathione peroxidase activities, reduced (GSH) and oxidized glutathione (GSSG) levels, and malondialdehyde contents were measured by a colorimetric method. The levels of intracellular reactive oxygen species and calcium were determined by flow cytometry. The results showed that SQS reduced LDH release and increased cell viability in a dose-dependent manner up to 10 microM and concomitantly decreased malondialdehyde and GSSG contents, while significantly increased GSH contents and the activities of superoxide dismutase, catalase and glutathione peroxidase. Moreover, treatment with SQS decreased intracellular reactive oxygen species levels and alleviated calcium accumulation in cardiomyocytes undergoing anoxia-reoxygenation. It is suggested that SQS could protect cardiomyocytes against oxidative stress induced by anoxia-reoxygenation by attenuating reactive oxygen species generation and increasing activities of endogenous antio

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